Degenerative process affecting articular cartilage of one or more joints, most commonly knee and hip. The spine is often involved, so be careful with neck position during intubation. Often due to wear and tear (think age, obesity). Minimal inflammatory reaction
Prevalent in the orthopedic population and may involve pulmonary (pleural effusion), cardiac (pericardial thickening and effusion, conduction defects, cardiac valve fibrosis), and musculoskeletal systems (usually multiple joints, symmetrical involvement). It differs from OA as it is an immune-mediated joint destruction due to inflammatory processes in the synovial membranes
Advanced RA patients are at high risk for compression of the spinal cord with intubation due to cervical instability, and some require pre-op X-rays. RA patients may be on steroids; note for perioperative steroid treatment, be aware of thin and atrophic skin, adrenal insufficiency, and impaired immune system
Both diseases can mask cardiac disease as patients have low exercise tolerance due to pain and joint restrictions
Regional advantages
Induced hypotension during surgery, keeping MAP >65 and/or within 20 to 30% of baseline MAP for HTN patients
Evaluate ongoing blood loss, note field bleeding, laps=100ml/ 4x4=10ml
One unit of RPBC increases Hct 2-3%/Hgb 1gm
Use of blood salvage techniques
Seen with multiple traumatic injuries and surgeries involving long bone fractures. Mortality 10-20%. Must have one major and a minimum of four minor symptoms
Male
Age 20-30 yrs
Hypovolemic shock
Intramedullary instrumentation
Rheumatoid arthritis
Total hip arthroplasty using cementing femoral stems
Bilateral knee surgery
Major
Minor
Nonspecific, only supportive (oxygenation, hydration, nutrition, prevention DVT/stress ulcer)
A significant cause of death after trauma/surgery to lower extremities. Evaluate risk factors (advanced age, prolonged immobility or bed rest, prior history, cancer, pre-existing hypercoagulable state)
Hip surgeries pose the most significant risk
Reduce surgical blood loss
Decreased blood in the field, which helps reduce operating time
Max 300 mmHg for UE and 500 mmHg for LE
Effects of tourniquet inflation: cellular hypoxia within 2 min, progressive cellular acidosis, an endothelial capillary leak develops after 2 hr, no somatosensory evoked potentials, and nerve conduction after 30 min
Tourniquet pain manifesting after 45-60 min, leading to increased HR and BP
Effects of tourniquet release: transient fall in core temp (0.6 °C for every hr), metabolic acidosis (increase K+, CO2, bicarbonate), fall in pulmonary and systemic arterial pressures, increase in ETCO2, release of acid metabolites (thromboxane - part of the lipid family eicosanoids) into the central circulation
Monitor the patient's temp via esophageal probe, skin, or F/C during the procedure
Use warming devices in the OR such as Forced air warmer and hot fluid lines. Cover the patient's head as much as possible (large area of heat loss)
Room temperature regulation as needed
Administer antibiotics within 60 min of incision (Vancomycin within 120 min)
Monitor regional sites for infection
Increased risk of injury with various positions requiring diligence by the providers
Pad pressure points (note ring under the ear, monitor pressure on the eye if lateral), ulnar nerve padding, prevent alopecia
Avoid hyperflexion of limbs/brachial plexus